From The TV Series “The Life Of Mammals”.

Your blog is super cool! I have a few questions. How do you get your equipment and chemicals to carry out your experiments? I was just wondering as I'd like to start doing experiments at home. What would be a good experiment to start with also? Sorry if you have already answered these questions

Hey, thanks for the kind words! This is going to be a long answer! Let’s start with equipment:

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New discovery could be a major advance for understanding neurological diseases

The discovery of a new mechanism that controls the way nerve cells in the brain communicate with each other to regulate our learning and long-term memory could have major benefits to understanding how the brain works and what goes wrong in neurodegenerative disorders such as epilepsy and dementia. The breakthrough, published in Nature Neuroscience, was made by scientists at the University of Bristol and the University of Central Lancashire.   The findings will have far-reaching implications in many aspects of neuroscience and understanding how the brain works.

The human brain contains around 100-billion nerve cells, each of which makes about 10,000 connections to other cells, called synapses. Synapses are constantly transmitting information to, and receiving information from other nerve cells. A process, called long-term potentiation (LTP), increases the strength of information flow across synapses. Lots of synapses communicating between different nerve cells form networks and LTP intensifies the connectivity of the cells in the network to make information transfer more efficient. This LTP mechanism is how the brain operates at the cellular level to allow us to learn and remember. However, when these processes go wrong they can lead to neurological and neurodegenerative disorders.

Precisely how LTP is initiated is a major question in neuroscience. Traditional LTP is regulated by the activation of special proteins at synapses called NMDA receptors. This study, by Professor Jeremy Henley and co-workers reports a new type of LTP that is controlled by kainate receptors.

This is an important advance as it highlights the flexibility in the way synapses are controlled and nerve cells communicate. This, in turn, raises the possibility of targeting this new pathway to develop therapeutic strategies for diseases like dementia, in which there is too little synaptic transmission and LTP, and epilepsy where there is too much inappropriate synaptic transmission and LTP.

Jeremy Henley, Professor of Molecular Neuroscience in the University’s School of Biochemistry in the Faculty of Biomedical Sciences, said: “These discoveries represent a significant advance and will have far-reaching implications for the understanding of memory, cognition, developmental plasticity and neuronal network formation and stabilisation. In summary, we believe that this is a groundbreaking study that opens new lines of inquiry which will increase understanding of the molecular details of synaptic function in health and disease.”

Dr Milos Petrovic, co-author of the study and Reader in Neuroscience at the University of Central Lancashire added: “Untangling the interactions between the signal receptors in the brain not only tells us more about the inner workings of a healthy brain, but also provides a practical insight into what happens when we form new memories. If we can preserve these signals it may help protect against brain diseases.

“This is certainly an extremely exciting discovery and something that could potentially impact the global population. We have discovered potential new drug targets that could help to cure the devastating consequences of dementias, such as Alzheimer’s disease. Collaborating with researchers across the world in order to identify new ways to fight disease like this is what world-class scientific research is all about, and we look forward to continuing our work in this area.”

May 18, 1969 — Inside mission control at the Johnson Space Center, Houston, during the first day of the Apollo 10 mission. (NASA)

Katie - Champ de Mars, Paris

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The Landscapes and Skylines of Howl’s Moving Castle ハウルの動く城 

What’s Up for July 2017

Prepare for the August total solar eclipse by observing the moon phases this month. Plus, two meteor showers peak at the end of July.

Solar eclipses occur when the new moon passes between the Earth and the sun and moon casts a traveling shadow on Earth. A total solar eclipse occurs when the new moon is in just the right position to completely cover the sun’s disk.

This will happen next month on August 21, when the new month completely blocks our view of the sun along a narrow path from Oregon to South Carolina.

It may even be dark enough during the eclipse to see some of the brighter stars and few planets!

Two weeks before or after a solar eclipse, there is often, but not always, a lunar eclipse. This happens because the full moon, the Earth and the sun will be lined up with Earth in the middle.

Beginning July 1, we can see all the moon’s phases.

Many of the Apollo landing sites are on the lit side of the first quarter moon. But to see these sites, you’ll have to rely on images for lunar orbiting spacecraft.

On July 9, the full moon rises at sunset and July 16 is the last quarter. The new moon begins on July 23 and is the phase we’ll look forward to in August, when it will give us the total solar eclipse. The month of July ends with a first quarter moon.

We’ll also have two meteor showers, both of which peak on July 30. The Delta Aquarids will have 25 meteors per hour between midnight and dawn.

The nearby slow and bright Alpha Capricornids per at 5 per hour and often produce fireballs.

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Tuz Gölü - Cereal  / WORDS & PHOTOS: Peter Edel

FOR THE AMERICAN COLOUR FIELD PAINTER BARNETT NEWMAN, THE EMPTY, BOUNDLESS LANDSCAPE ENHANCED AN INDIVIDUAL’S SENSE OF PRESENCE WITHIN THEM. THE TUZ GÖLÜ, THE SALT LAKE LOCATED IN THE CORE OF TURKEY’S ANATOLIAN PENINSULA, IS ONE OF THE PLACES IN THE WORLD WHERE THIS UNDERSTANDING IS EXPERIENCED MOST PROFOUNDLY.

Theatre time. All dancer have their own ways of getting ready for a show. I believe that a consistent routine is important to preparing for what’s ahead in a few hours. Because Forsythe’s “Artifact” is so hard on the body and I’m in every show, I tend to get to the theatre pretty early to make sure everything is ready, to put on some “normatec” boots (a compression boot for athletes that helps greatly with fatigue) and do hair and makeup. - Lia Cirio

Lia Cirio - Boston Opera House

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The Science of Smog

On July 26, 1943, Los Angeles was blanketed by a thick gas that stung people’s eyes and blocked out the Sun. Panicked residents believed their city had been attacked using chemical warfare. But the cloud wasn’t an act of war. It was smog. A portmanteau of smoke and fog, the word smog was coined at the beginning of the 20th century to describe the thick gray haze that covered cities such as London, Glasgow, and Edinburgh.

This industrial smog was known to form when smoke from coal-burning home stoves and factories combined with moisture in the air. But the smog behind the LA panic was different. It was yellowish with a chemical odor. Since the city didn’t burn much coal, its cause would remain a mystery until a chemist named Arie Haagen-Smit identified two culprits, volatile organic compounds, or VOCs, and nitrous oxides. VOCs are compounds that easily become vapors and may contain elements, such as carbon, oxygen, hydrogen, chlorine, and sulfur. Some are naturally produced by plants and animals, but others come from manmade sources, like solvents, paints, glues, and petroleum. Meanwhile, the incomplete combustion of gas in motor vehicles  releases nitrous oxide. That’s what gives this type of smog its yellowish color.

VOCs and nitrous oxide react with sunlight to produce secondary pollutants called PANs and tropospheric, or ground level, ozone. PANs and ozone cause eye irritation and damage lung tissue. Both are key ingredients in photochemical smog, which is what had been plaguing LA. 

Smog isn’t just an aesthetic eyesore. The two forms of smog irritate the eyes, nose, and throat, exacerbate conditions like asthma and emphysema, and increase the risk of respiratory infections like bronchitis. Smog can be especially harmful to young children and older people and exposure in pregnant women has been linked to low birth weight and potential birth defects. Secondary pollutants found in photochemical smog can damage and weaken crops and decrease yield, making them more susceptible to insects.

After the Great Smog of London shut down all transportation in the city for days and caused more than 4,000 respiratory deaths, the Clean Air Act of 1956 banned burning coal in certain areas of the city, leading to a massive reduction in smog. Similarly, regulations on vehicle emissions and gas content in the US reduced the volatile compounds in the air and smog levels along with them. 

Smog remains a major problem around the world. Countries like China and Poland that depend on coal for energy experience high levels of industrial smog. Photochemical smog and airborne particles from vehicle emissions affect many rapidly developing cities, from Mexico City and Santiago to New Delhi and Tehran. Governments have tried many methods to tackle it, such as banning cars from driving for days at a time. As more than half of the world’s population crowds into cities, considering a shift to mass transit and away from fossil fuels may allow us to breathe easier.

From the TED-Ed Lesson The science of smog - Kim Preshoff

Animation by Juan M. Urbina Studios